Dht derived steroids list

Hi Ali: Regarding the mint studies…I just saw this on Suppversity…regarding the difference between mints effects on male and female rats…..Spearmint (M. spicata): At least in women spearmint tea has been shown to increase estrogen and luteinizing hormone in the follicular phase of their menstrual cycle (Aktodgan. 2007). In a 2004 study that was conducted on male rodents, on the other hand, the daily administration of peppermint tea (M. spicata) for a period of 30days lead to significant increases in luteinizing and follicle stimulating hormone and increases in serum testosterone, yet with the serious downside of “extensive degenerative changes in the germinal epithelium and spermatogenesis arrest compared with the findings in the testicular biopsies of the control group” (Aktogan. 2003) interesting for sure!

However, we know very little about Omega-6 and the ‘pro-inflammatory’ eicosanoids, and more specifically when the group 2 prostaglandins are a bad inflammatory, or a good inflammatory. Obviously, we need inflammatory processes, it’s only when we enter a chronic inflammatory state, that these are potentially bad news. Then of course, we have Swiss Temple’s theory, which overcame an evil group 2 prostaglandin, with another pro-inflammatory prostaglandin (a vast oversimplification I know, but that’s the general gist). So the story is far from simple with a variety of feedforward and feedback loops within the Group 2 eicosanoids, interleukins etc then there are crossed effects from the group 1 and Group 3 eicosanoids, which are typically inhibitory to the Group 2s, and countless other interactions with other fatty acids, let alone other molecules found in each cell nucleus. So it certainly isn’t out of the realm of possibilities…..

There is a myth being spread around both the net and the magazines today regarding which steroids are more likely to cause hair loss, but before we get to that, we must first gain an understanding of the subject of steroid “families”. Every AAS sold on the market today technically belongs to one of three classes (or families) of steroids. These are the testosterone-based, 19 nor-based, and DHT-based families of steroids. Each AAS is defined as belonging to one of these three classes of steroids, based on which of the three molecules it was originally derived from. For example, since the steroid Anavar is an alteration of the DHT molecule, it is classified as belonging o the DHT family of steroids. Since Dianabol is an alteration of the testosterone molecule, it is classified as belonging to the testosterone family of steroids. In reality, “all” steroids are derived from the testosterone molecule itself, but we utilize these 3 classes of AAS as basic sub-categories, in order to further categorize steroids into more specific groups.

Over-inhibition of 5AR results in increased availability of testosterone, which may be shunted, via aromatase, to estradiol, causing “feminization,” ., gynecomastia, one of the side effects of 5AR inhibitor drugs; and decreased production of not only 5α-DHT, but its metabolites, 5α-Androstane-3α-17β-diol ( aka 3α-adiol, a storage form of 5α-DHT), and 5α-Androstane-3β-17β-diol ( aka 3β-adiol), an estrogen receptor beta (ERβ) ligand that promotes normal cellular differentiation, thus lessening risk of benign prostatic hypertrophy and prostate cancer. 22 , 23 ,  24

Dht derived steroids list

dht derived steroids list

Over-inhibition of 5AR results in increased availability of testosterone, which may be shunted, via aromatase, to estradiol, causing “feminization,” ., gynecomastia, one of the side effects of 5AR inhibitor drugs; and decreased production of not only 5α-DHT, but its metabolites, 5α-Androstane-3α-17β-diol ( aka 3α-adiol, a storage form of 5α-DHT), and 5α-Androstane-3β-17β-diol ( aka 3β-adiol), an estrogen receptor beta (ERβ) ligand that promotes normal cellular differentiation, thus lessening risk of benign prostatic hypertrophy and prostate cancer. 22 , 23 ,  24

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